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Cancer Miracle – so simple!

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Cancer Miracle – so simple!

This video link says it all – do take a moment to watch and put on the back burner just in case :

 

Further research can be found here:

 

 

Coconut Oil Alternative Cancer Treatment: Your Complete Guide

Lauric acid, found in coconuts, is already a well-established destroyer of bacteria, viruses, fungi, and parasites, but recently scientists have discovering something even more amazing: it can also kill cancer cells. About 60% of coconut oil is comprised of lauric acid.  In fact, coconut oil with it’s potent anti-viral and anti-bacterial properties, contains more lauric … Continue readingCoconut Oil Alternative Cancer Treatment: Your Complete Guide

 

 

This One Oil Knocks Out Cancer Cells Like Crazy!

One of the most common cancers, affecting both men and women, is colon cancer. There have been a good number of natural remedies and foods that have been linked to prevention, but there have not been many studies which show that these remedies kill cancer cells. This is a common theme in the mainstream medical … Continue readingThis One Oil Knocks Out Cancer Cells Like Crazy!

 

The lauric acid-activated signaling prompts apoptosis in cancer cells

https://www.nature.com/articles/cddiscovery201763

The lauric acid-activated signaling prompts apoptosis in cancer cells

Cell Death Discovery volume 3, Article number: 17063 (2017) 

Abstract

The saturated medium-chain fatty-acid lauric acid (LA) has been associated to certain health-promoting benefits of coconut oil intake, including the improvement of the quality of life in breast cancer patients during chemotherapy. As it concerns the potential to hamper tumor growth, LA was shown to elicit inhibitory effects only in colon cancer cells. Here, we provide novel insights regarding the molecular mechanisms through which LA triggers antiproliferative and pro-apoptotic effects in both breast and endometrial cancer cells. In particular, our results demonstrate that LA increases reactive oxygen species levels, stimulates the phosphorylation of EGFR, ERK and c-Jun and induces the expression of c-fos. In addition, our data evidence that LA via the Rho-associated kinase-mediated pathway promotes stress fiber formation, which exerts a main role in the morphological changes associated with apoptotic cell death. Next, we found that the increase of p21Cip1/WAF1 expression, which occurs upon LA exposure in a p53-independent manner, is involved in the apoptotic effects prompted by LA in both breast and endometrial cancer cells. Collectively, our findings may pave the way to better understand the anticancer action of LA, although additional studies are warranted to further corroborate its usefulness in more comprehensive therapeutic approaches.

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